You’re on the right page if you are looking to download free Robbins Basic Pathology 9th Edition with an easy and direct download link. This free pathology robbins latest edition deals with Cell Injury, Cell Death, and Adaptations, Inflammation and Repair, Hemodynamic Disorders, Thromboembolism, and many other topics in detail. This Free PDF Medical pathology knowledge you need, the way you need it, from the name you can trust. The unbeatable team of authors of this medical textbook helps you efficiently master the basics you need to know for your USMLE courses and exams.
This is an exciting time for medical students because the fundamental mechanisms of the disease are being revealed. at an impressive rate. Pathology is critical to understanding the molecular basis of disease, and we have attempted to capture the essence of this new knowledge in Robbins’ Ninth Edition of Basic Pathology. We firmly believe that pathology forms the scientific basis of medicine and advances in the basic sciences ultimately help us to understand disease in the individual patient. So while many of the new discoveries in genomics and personalized medicine are covered in the initial chapters on general pathology, we have endeavoured to include the impact of scientific advances on diseases of the organ systems described throughout the text. To emphasize the importance of disease mechanisms in the practice of medicine, we have presented sections dealing with pathogenesis. In recent years, understanding of the molecular basis of the disease has led to the development of “targeted therapies.” Do they stand out in the form of “Targeted Therapy” graphics?
|Book Name||Robbins Basic Pathology|
|Author of Book||Vinay Kumar, Abul K. Abbas , Jon C. Aster|
This Pocket Companion to the robbins 9th edition of Pathology has the given below key features in this version.
- Gain a broad understanding of all essential pathology concepts with the expert guidance of an all-star editorial team.
- Understand the connections between basic science and clinical medicine with clinicopathological correlations throughout.
- Access information anywhere, from the cafeteria to the classroom, with full-text online access at studentconsult.com.
- Take your learning further with targeted therapy boxes, clinical cases, virtual microscope slides, and online self-assessment questions.
Coming to the 40th year of the publication of Robbins Basic Pathology, it is useful to quote Stanley Robbins from the Preface to the first edition (1971): “Of both books and men, it can be seen that the fat contains the thin. struggling to get out. In a sense, this book has such a relationship with its more substantial progenitor, Robbins Pathology. It arose out of an appreciation of the dilemma of the modern medical student. As the curriculum has been restructured to place greater emphasis on clinical experience, the time for reading is shortened accordingly. As of this writing, rare and esoteric injuries are unapologetically omitted, infrequent or trivial ones described only briefly. However, we found it important to consider the main disease entities in considerable detail. “The goals of this issue of“ Baby Robbins ”remain true to this vision of Stanley Robbins.
Realizing that the modern medical student is overwhelmed trying to synthesize the essentials with the “state of the art,” we have continued to use summary charts designed to provide students with key “take-home” messages. . These have been withheld at the risk of adding a few extra pages to the book, as students have consistently told us that they find them useful. Many new pieces of four-colour art (schematics, flow charts, and schematic representations of disease) have been added to facilitate understanding of difficult concepts such as cell cycle control, cancer gene functions, HIV interactions with its receivers. and the biochemical basis of apoptotic cell death. More illustrations have been added, bringing the total to over 1,000.
The table format and colour palettes have been changed for clarity. Despite extensive changes and revisions, our goals remain essentially unchanged. Although we have entered the genomic era, traditional macroscopic and microscopic analysis tools are still useful and morphological changes are highlighted for quick reference. The strong emphasis on clinicopathological correlations is maintained and, wherever understood, the impact of molecular pathology on the practice of medicine is emphasized. We are pleased that all of this has been accomplished without any “bulge” at the waistline of the text. We continue to firmly believe that clarity of writing and proper use of language enhance understanding and facilitate the learning process. Generations of students have told us that they enjoy reading this book. We hope this edition is worthy and possibly enhances the tradition of your ancestors.
Topics of this Edition:
Robbins Basic Pathology 9th Edition PDF Free has the following topics
1 Cell Injury, Death and Adaptation
2 Inflammation and Repair
3 Hemodynamic Disorders, Thromboembolism, and Shock
4 Diseases of the Immune System
6 Genetic and Pediatric Diseases
7 Environmental and Nutritional Diseases
8 General Pathology of Infectious Diseases
9 Blood Vessels
11 Hematopoietic and Lymphoid System
13 Kidney and Its Collecting System
14 Oral Cavity and Gastrointestinal Tract
15 Liver, Gallbladder, and Biliary Tract
17 Male Genital System and Lower Urinary Tract
18 Female Genital System and Breast
19 Endocrine System
20 Bones, Joints and Soft Tissue Tumors
21 Peripheral Nerves and Muscle
22 Central Nervous System
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Introduction of Pathology:
Literally translated, pathology is the study (logos) of disease (pathos, suffering). It involves the investigation of the causes of the disease and the associated changes at the cell, tissue and organ level, which in turn give rise to the presenting signs and symptoms of the patient. There are two important terms that students will encounter throughout their study of pathology and medicine:
- Etiology is the origin of a disease, including underlying causes and modifying factors. It is now clear that the most common diseases, such as hypertension, diabetes, and cancer, are caused by a combination of inherited genetic susceptibility and various environmental triggers. Understanding the genetic and environmental factors that underlie disease is an important topic in modern medicine.
- Pathogenesis refers to the steps in the development of a disease. It describes how etiological factors trigger cellular and molecular changes that give rise to the specific functional and structural abnormalities that characterize the disease. While etiology refers to why a disease arises, pathogenesis describes how a disease develops.
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Peripheral Nerves and Muscles:
The major components of the neuromuscular system, the peripheral nerves and skeletal muscles, act as both effectors and sensors for the central nervous system, and in doing so allow thought and sensation to give rise to physical actions and cognitive responses. The principal component of the motor system is the motor unit, which is composed of one lower motor neuron and its associated peripheral axon, neuromuscular junctions, and innervated skeletal muscle fibers. Both the anatomic distribution of lesions and specific signs and symptoms are helpful in classifying neuromuscular diseases and in distinguishing them from diseases of the central nervous system. Accordingly, the discussion of neuromuscular disorders is organized along anatomic lines, highlighting the clinical features that are most useful in their diagnosis.
DISORDERS OF PERIPHERAL NERVES
The two major functional elements of peripheral nerves are axonal processes and their myelin sheaths, which are made by Schwann cells. Axonal diameter and myelin thickness are correlated with each other and with conduction velocity; they can be used to distinguish among different types of axons, which mediate distinct sensory modalities and motor function. Light touch, for example, is transmitted by thickly myelinated large-diameter axons with fast conduction velocities, while temperature sensation is transmitted by slow, unmyelinated thin axons. In the case of myelinated axons, one Schwann cell makes and maintains exactly one myelin segment, or internode, along a single axon
Patterns of Peripheral Nerve Injury
Most peripheral neuropathies can be subclassified as either axonal or demyelinating, even though some diseases exhibit mixed features. Axonal neuropathies are caused by insults that directly injure the axon. The entire distal portion of an affected axon degenerates. Axonal degeneration is associated with secondary myelin loss (Fig. 21–1, B), a process sometimes referred to as Wallerian degeneration. Regeneration takes place through axonal regrowth and subsequent remyelination of the distal axon (Fig. 21–1, C). The morphologic hallmark of axonal neuropathies is a decrease in the density of axons, which in electrophysiologic studies correlates with a decrease in the strength of amplitude of nerve impulses.
Demyelinating neuropathies are characterized by damage to Schwann cells or myelin with relative axonal sparing, resulting in abnormally slow nerve conduction velocities. Demyelination typically occurs in individual myelin internodes randomly; this process is termed segmental demyelination (Fig. 21–1, B). Morphologically, demyelinating neuropathies show a relatively normal density of axons and features of segmental demyelination and repair. This is recognized by the presence of axons with abnormally thin myelin sheaths and short internodes (Fig. 21–1, C). These latter changes are best demonstrated on teased fiber preparations, which allow the examination of several adjacent myelin internodes along a segment of an individual axon (described later)
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